Molar Mass of Oxygen Essay Example for Free

Molar Mass of Oxygen Essay Applying the individual % of elements in Water to the molar mass Conclusion____________________________________________________________ In conclusion the experimental value of the molar mass of oxygen is. To achieve this result, we wrote the chemical reaction, compared the ratio of mole between and, find the mole produced by water using the formula, calculate the molar mass from the mol value, calculate the % of individual elements in water, and finding oxygens molar mass from the overall molar mass. Analysis Evaluation ___________________________________________________ -In analysis, our results show an unreasonable result. The majority of the failure is from the volume result. From Avogrados law we know that. As such, because the average volume (0.051 L) was low, the mol resulting from it is also low. A small mol results in a large Mr value, or in our case the Mr of Oxygen is too high. -Another point to that is that if the experiment was assuming r.t.p, our results prove to be incorrect again. If we compared the mol, the actual mol results of andusing the ideal gas laws is different than if we calculated using normal Stoichiometry. From Stoichiometry, the actual mol of is, and if we compared it to a ratio of 2:2, the actual mol value of is also 0.138 mol. However in our experiment using the gas law, our mol result was. Again this is due to the fact of the low amount of volume recorded. -In addition to that point, from the section to: Calculate molar mass of Water using the mass of Hydrogen Peroxide , another flaw to the experiment was the amount of uncertainty in the result. From the section we apply the law of conservation of mass stating that no mass is created or destroyed. Or in this experiments case, most of the mass decomposes into Oxygen gas and finally results in the volume of water displaced from the process. However as shown from the low amount of volume our group collected, this can result in two conclusions. The first conclusion is that most of the oxygen gas that eventually turns into the volume of water left is lost to the surroundings (from the test tube openings) OR the second conclusion is that our group did not wait long enough and thus lost some potential volume results. -To evaluate, the major flaw to the experiment was the value of the data collected. Part of the reason was because we used Vaseline as a tool to reduce the amount of energy (in the form of the Oxygen gas) to the surroundings. Vaseline is a good measure to do so, however a better alternative is probably to increase the number of trials, to have a more close system that may significantly reduce the uncertainties, and to maybe reduce the length of rubber tube (by reducing the length of rubber tube the amount of water left is reduced). References IB- Foundation books. Raymond Chang (pg: 87, 181, 182, 191.etc)

The unemployment problem and possible solutions

The unemployment problem and possible solutions Unemployment is a residual and individual problem which effect on whole economy. By late the 1980s unemployment becomes a big policy and public consensus for the G5 countries (UK, USA, Japan, France and Germany). It continued argue that among the G5 countries the level of UK economy was an economy of Unemployment with all the human and economic waste and inefficiency such as an economy entails. In view of the rapid rise in unemployment that has occurred between 1990 and 1991. But recently the UK has lower unemployment rates than France and Germany. However a number of logical thought trying to explain the policy of unemployment rates and address the solution of Unemployment. Unemployment Unemployment defined as the numbers of people of working age who are able and available for work at current wage rates and who do not have a job. But the unemployment rates in the proportion of unemployed people in the economically active in labour force. Unemployment Rates calculated by: number of unemployed/number of economical active X100.There is two basic ways government can measure the unemployment rates such as The Claimant Count: means the number of people when claiming their unemployment benefit from government on any given period time. Labour Force Surveys: by this survey government count number of employed including number of unemployed people. According to Labour Force survey the graph shows that in 1990 unemployment rates fall at just over 6% where employment rates picked at just under 76%. Currently (source: IMF) UK unemployment rates more than 8% whereas Germany holds well above 10% unemployment rates. Zero Unemployment Rates It is true that the economy unemployment rate could not at zero if an economist full employment because there are two type of unemployment exist in our economy such as: Structural Unemployment: It occurs when consumer demand for the new product. For example when new technological progress has made skills blockage such as computer introduced white collar disappeared. Frictional Unemployment: It occurs when for the particular skill demand for labour and supply for labour are not match or people are not aware for the job opportunity or geographical match of workers ongoing process. Besides those reason unemployment could not zero for some institutional phenomenon such as: Minimum wage law may make it too expensive to hire a extra labour Government employment benefit reduce job interest Government restriction on institution may reduce job arability. Racism or gender discrimination may decrease interest of job. etc However there are numbers of principal schools of thought in macroeconomics offered the cause of unemployment such as Classical Thought Keyness Thought Neo -Classical Thought Monetarism Thought New classical Thought New Keynesian Thought Classical Thought The classical thought assumed that the economy would tend to full equilibrium if left its own. According classical theory, labour market operated demand of labour and supply of labour when balanced by price signals. From the graph shown that there is unemployment exist when excess labour supply (N2) and demand of labour (N3). The classical school of thought explain that if excess labour supply existence in economy, wages would fall(W1 to W*) until the labour market clearing equilibrium is restore, alternatively when excess labour demand existence labour shortage would push up wages and restore the equilibrium(NFull). Says Law is justified the classical view and law said that: supply creates its own demand. That means the economy is in a permanent state of full-employment equilibrium. Because says law guarantees any increase in output of goods and services will sold for sufficient demand and therefore firm will never reduce output or cut the jobs. However, if there is unemployment, market forces should quickly eliminate it and restore equilibrium. But after 1929-33 great depression the whole world economy collapsed in industrial capitalism and the classical school of theory could not explain the established economic wisdom. This depression eventually gave to raise Keynesian thought. Keynesian Thought Keynes his most famous work The General Theory of Employment, Interest and Money (1936) argued that could not settle at Equilibrium and it would not change the labour market situation because of aggregate demand. If aggregate demand fell, output and employment could fall and the economy could become trapped in a less than full-employment equilibrium. The graph shows that when demand for labour fall (D1 to D2) the wage also fall (W to W1) and unemployment would formed (ab). Nevertheless 1970s continuous unemployment and inflection failed the Keynesians demand deficient unemployment and this argument considered as Keynesians two analytical frame works such as the 45 degree model and the Philips curve which is known as a Neo- classical theory. Neo Classical Thought According the neo classical thought the 45 degree demonstrates the Keynesian aggregate demand .It means to using the appropriate fiscal policy if aggregate demand add and reduce the economy fall in inflection pressure. More precisely where output and employment are below their full employment level if government cut tax or higher government expenditure which is increases the aggregate demand. On the other hand where output and employment are at their full employment level if government increase tax or decrease government expenditure in this situation demand- pull inflationary pressures are exists. Therefore the aggregate demand management will maintain the economy at close to full employment equilibrium both unemployment and inflection need be a problem. The graphs show that if aggregate demand fall a positive demand shock occurred at full employment equilibrium position (Ye) and meanwhile inflationary gap existed in economy. Conversely the 45 degree aggregate demand says that the unemployment and inflection not appeared in same time. In the late 1950s Philips curve more purify the Keynesian thought. In 1958 Professor A.W.Philips illustrated a statistical relationship between unemployment and inflection The Philips curve shows the inverse relationship between unemployment rates and inflection. It argued that if government wants to reduce unemployment it has to accept higher inflation as a trade off. The graphs shows that if unemployment rates fall (1.5% to 1%) inflation rates up (2 to 4%)S Although in 1970s the Philips curve was unable to explain the problem of unemployment and inflection which is going up together stagflation. In mean while time two economists Milton Friedman and Edwards Phelps appeared with monetarism theory that able to show concurrently inflection and unemployment based on expectations augmented Philips curve. Monetarism Professor Friedman argued that there were a series of different Philips curve for each level of expected inflection. He persuaded that when government injects resources into the economy once again the unemployment fall in short-term but there would occurred high inflation. As a result people expected inflection to occur then they would anticipate and expected a correspondingly higher wage rise. The graph revealed that unemployment below Un to U* the series of Philips curve (SRPC1) moved alone with inflation rates zero to 4% because the expectation has been changed and people adept new rates. But Friedman failed to long term unemployment related with inflation rates which is elaborated by New Classical viewed. New Classical Thought Robert Locus who is one of the new classical economist argued that announced and unannounced fiscal and monetary policy are affected on out and employment because of natural rate of unemployment will alter the equilibrium and this thought actually expand the rational expectation. The graph shows without short term reduction of unemployment (Un to U*) rational agent would anticipated an inflationary environment. New Keynesian Thought The new Keynesian thought wrecked the long run Philips curve which is breakdown by Friedman depends on NAIRU (Non Accelerating inflation rates of unemployment). In Behavioural theory George Akerlof argued at the low level of inflation permanent trade off between inflation and unemployment because low inflation not silent. In the diagram shows when unemployment fall from U a silent inflation becomes higher. Furthermore according New Keynesian point of view real wage rate could establish long unemployment equilibrium. The diagram exposed that a higher market clearing efficiency wage paid consequent unemployment (N2-N1) whereas aggregated demand shock shifting the labour demand curve which is lead the unemployment ( N2 to N3). Most Satisfactory Explanation on nature of European Unemployment The classical thought believed that supply automatically creates full employment and efficient market economy drive the unemployment problem where a little need of government interference. But 1929-33 great depression pushed the European unemployment rates to unprecedented level and whole economy collapsed. Country 1921-29 1930-38 United States 7.9 26.1 United Kingdom 12 15.4 France 3.8 10.2 Germany 9.2 21.8 The table shows the percentage of unemployment rates of European countries during depression period. In 1930-38 USA enjoyed high Unemployed rates 26.1% The depression eventually gave to raise the Cambridge economist John Maynard Keynes thought and he identified the root of the problem as a lack of aggregated demand. He explained that if aggregate demand fall the economy were hit by adverse shocks which create a fall the business confidence where Says law would failed to hold as firms cut investment, output and employment and this process could leave the economy in less than full unemployment equilibrium. Moreover a less than full-employment output would find just enough demand for that output and the economy would be stuck in a slump. Keynes suggested that government attempt stabilized the policy for settle the level of output and full employment. Despite the fact that until 1970s the Keynesian aggregate demand management dominated western policymaking the economy in of overheating and facing the inflationary pressured. In1970s inflation rates increase 10% to more the 20%. The graph shows that after 1970 inflation rates in UK and Japan reached at well above 20% whereas USA and France more than 15% abut Germany enjoyed the less inflationary rates. In the mean while time G5 countries were suffered by high unemployment. From the following graphs we can see that in 1970s unemployment rates increased rapidly where full employment appear only 2.5% . In 1970s economy are experienced by rising unemployment and inflation which made together stagflation where Keynesian policy failed to explain the new dilemma. On the other hand Keynesian appeared that most unemployment arise outside labour market but the great depression and early 1980s and 1990s recession Keynesian view unsuccessful to explain Frictional and Structural unemployment. Conversely Friedman views clear the all of Keynesians confusion which is based on expectations augmented Philips curve. Because 1970s stagflation redundant the Philips curve. Whereas Friedman indentified that the cause of inflation is balanced by the natural rate of unemployment and this unemployment occurred inside labour market which should be in microeconomic nature, cause macroeconomic policy not affective in the long run. He also argued the stabilization policy which was driven the Post war boom means it ineffective to maintain the economics at potential GDP and full employments and it should be d estabilized cause economic will stable inherently. Yet Monetarist assumption abandoned the1980s and 1990s recession and in monetarist view labour market are not flexible even though the lack of competitiveness has obsessed the real wage. After all in my point of view the Keynesian school thought likely approach than other school of thought though monetarist would favour to abandon the stagflation. But if we see the 1970s, 1980s and 1990s inflation and unemployment is high but overall GDP rate remarkable. In addition Keynesian view is applicable in recent recession. Recently viewed that the Europe countries aggregated demand fall and lower rates of growth people are less interest to consume which tend to increased unemployment hand .Besides the Keynesian thought not decline fiscal and monetary policy and Labour union power to determined the wage and right. Recent Unemployment and Solution In UK jobless jumped by 43,000 and unemployment reached at 8%. According to IMF survey global financial crisis impact on European output and employment and it increased the recent unemployment. In Germany, UK output falls in significantly which reduced the growth of employment. Moreover labour market flexibility, mainly the higher level of employment protection lean to reduced employment inflow and outflow and declines the labour reallocation. Further more rapid rise of structural unemployment, financial institutions collapse, cutting hours, early retirement tend to increased the Unemployment On the contrary mix labour market policies and flexibilities at firm level gradually employment has adjusted in UK and Germany. It is true that time accounts smooth the Germanys employment whereas government subsides decreased the working time. But wage flexibility and government support help to surpass the UK unemployment. Conclusion As final point unemployment is a major problem in the world economy. It is very difficult to bring down equilibrium position if it is not stop to increase at the first place and in the long run unemployed not able to participate in labour market. Therefore, government should initiate the improving labour market by increasing work incentives, reforming the operation house market and trade union.

Regulation of Gastric Acid Secretion

Regulation of Gastric Acid Secretion The stomach is a J-shaped enlargement of the gastrointestinal tract just lies below the diaphragm; the digestive tube dilates into an elongated pouchlike structure, the size of which varies according to several factors, notably the amount of distention. For sometime after a meal, the stomach is enlarged because of distention of its walls, but, as food leaves, the walls partially collapse, leaving the organ about the size of a large sausage. In adults the stomach usually holds a volume upto 1.0 to 1.5L. ANATOMY OF STOMACH 1.1.1 Divisions of the stomach: The fundus, body and the pylorus are the three divisions of the stomach. The fundus is the enlargement portion of the left and above opening of the oesophagus into the stomach. The body is the central part of the stomach, and the pylorus is its lower portion. 1.1.2 Curves of the stomach: The curve formed by the upper right surface of the stomach is known as the lesser curvature; the curve formed by the lower left surface is known as the greater curvature. 1.1.3 Sphincter muscles: Sphincter muscles guard both stomach openings. A sphincter muscle consists of circular fibres so arranged that there is an opening in the centre of them (like the hole in a doughnut) when they are relaxed and no opening when they are contracted. The cardiac sphincter controls the opening of the oesophagus into the stomach, and the pyloric sphincter controls the opening from the pyloric portion of the stomach into the first part of the small intestine. 1.1.4 Stomach wall: Gastric mucosa The epithelial lining of the stomach is thrown into folds, called rugae, and marked by depressions called gastric pits. Numerous coiled tubular- type glands, gastric glands, are found below the level of the pits, particularly in the fundus and body of the stomach. The glands secrete most of the gastric juice, a mucous fluid containing digestive enzymes and hydrochloric acid. 1.1.5 Functions of the stomach: The stomach carries on the following the functions: It serves as a reservoir, storing food until it can be partially digested and moved further along the gastrointestinal tract. It secretes gastric juice, containing acid and enzymes, to aid in the digestion of food. It carries on the limited amount of absorption of some water, alcohol, and certain drugs. It produces the hormone gastrin, which helps regulation of digestive functions. It helps to protect the body by destroying pathogenic bacteria swallowed with food or with mucous from the respiratory tract. 1.2 Regulation of gastric acid secretion: The mechanisms operating at the gastric parietal cells as summarized in the above figure. The terminal enzyme H+K+ATPase (proton pump) which secretes H+ ions in the apical canaliculi of parietal cells can be activated by histamine, Ach and gastrin acting via their own receptors located on the basolateral membrane of these cells.Out of the three physiological secretagogues, histamine, acting through H2 receptors, plays the dominant role, because the other two, gastrin and Ach act partly directly by releasing histamine from paracrine enterochromaffin like cells called histaminocytes located in the oxyntic glands. While H2 receptors activate H+K+ATPase by generating cAMP, muscarinic and gastrin receptors appear to function through the phospholipaseC -IP3-DAG pathway that mobilizes intracellular Ca+. The cAMP mediated proton pump activation also involves Ca+. The secretomotor response to gastrin and cholinergic agonists is expressed fully only in the presence of cAMP generated by H2 acti vation. As such, histamine participates in the acid response to gastrin and Ach at more than one levels, and H2 antagonists suppress not only histamine but also Ach, pentagastrin and in fact any gastric acid secretory stimulus. Gastrin is secreted from the antrum in response to rise in antral pH, food constituents and vagally mediated reflexes. The dominant muscarinic receptor mediating vagal responses is of M1 subtype. Its location on the ganglion cells of the intramural plexuses has been confirmed. The parietal cell muscarinic receptor is of the M2 subtype but the subtype of muscarinic receptor on histaminocytes has not been defined. Vagus releases Ach in dose proximity to histaminocytes and gastrin secreting cells, but apparently at a distance from the parietal cells. Prostaglandins have been ascribed a cytoprotective role in the gastric mucosa by augmenting mucus and bicarbonate secretion, as well as other actions. PGE2, produced by gastric mucosa, inhibits acid secretion by opposing cAMP generation and gastrin release. CONTROL OF GASTRIC ACID IN STOMACH: 1.3 Peptic Ulcer Peptic ulcers disease refers to a group of disorders characterized by circumscribed lesions of the mucosa of the upper gastrointestinal tract (especially of the stomach and duodenum). The lesions occur in regions exposed to gastric juices. When the stomachs natural protections from acid stop working ulcers will occur. Duodenal ulcers almost always develop in the duodenal bulb (the first few centimetres of the duodenum). A few, however, arise between the bulb and the ampulla. Gastric ulcers form most commonly in the antrum or at the antral-fundal junction. Nearly 80 % of peptic ulcers are duodenal the others are gastric ulcers. Most duodenal ulcers appear in people between ages 20 and 50 years, while gastric ulcer usually occurs between ages 45 and 55 years. Duodenal ulcer is twice as common in men as in women and gastric ulcers affect men and women equally. Approximately 10 to 20 % of gastric ulcer patients also have a concurrent duodenal ulcer. Gastric ulcer is often a chronic disease and may persist for 10 to 20 years characterized by repeated episodes of healing and re-exacerbation. Peptic ulcers occur when there is an imbalance between offensive factors and defensive mucosal factors (Goel and Bhattacharya, 1991). Ulceration in the mucosa can be because of either breakdown of mucosa with the development of surface defects or failure of restitution of mucosal integrity resulting in retardation or failure of healing of the ulcers. No apparent causal factor is sufficiently uniquely associated with peptic ulcers to warrant unequivocal implication in pathogenesis of the ulceration. The mechanism of defensive action consists of humoral, functional and neuronal factors. All these factors are responsible for the mucosal protection. The precise biochemical changes during ulcer generation are not clear yet, although various hypotheses have been proposed from time to time. Increased gastric motility, vagal over activity, mast cell degranulation decreased gastric mucosal blood flow and decreased prostaglandin level during stress condition is thought to be involved in ulcer generation. Similarly role of oxygen derived free radicals have been shown to play a role in experimental gastric damage induced by ischemia and reperfusion, hemorrhagic shock and ethanol administration. Helicobacter pylori a pathogen is now known to be the most common and important causes of gastric ulcer in humans (Davies et al., 1994), exhibits active inflammation with epithelial damage accompanied by neutrophil migration. Although the currently used drugs for ulceration are broadly classified into two, those that decrease or counter increase in acid-pepsin secretion and those that afford cytoprotection by virtue of their effects on mucosal defensive factors. Ulcer treatment can be carried out by reducing the action of aggravating factors. Since gastric acid is one of the major aggressive factor contributing to peptic ulcer disease, the reduction of gastric acid either by surgical or pharmacological intervention has been used to promote ulcer healing. However, not all patients, with gastric or duodenal ulcer have high acid secretion. In fact, only 30%-40% of cases with duodenal ulcer have hypersecretion of gastric acid and, in patients with gastric ulcer, acid secretion is either normal or low (Gupta et al., 1980). In these cases, decreased mucosal resistance might be the dominant factor. The neutralization of gastric acid can be done by antacid administration, but their effectiveness is only for a brief period. Muscarinic antagonists such as pirenzepine or telenzepine are effective inhibitors of acid production. The histamine H2-receptor antagonists (H2RA) like cimetidine, ranitidine, famotidine etc. act as potent inhibitors of acid secretion. Complete inhibition of parietal cells acid secretion by receptor antagonist is difficult because of complexity of known receptors on parietal cells and a variety of second messenger signaling system coupled to these receptors, which involve adenylate cyclase coupled with histamine receptor and intracellular Ca+2 with acetylcholine receptors. Thus, the most successful and desirable therapy is to inhibit the enzyme responsible for acid secretion. Moreover, H2RA have been reported to have some adverse reactions ranging from gastric carcinoid to tolerance and rebound acid secretion. Inhibition of gastric H+/K+ -ATPase of the pariet al cell by drugs such as by proton pump inhibitors (PPIs) like omeprazole, lansoprazole, timoprazole, etc. has been shown to be effective in the treatment of peptic ulcer disease. However, such agents irreversibly inactivate the ATPase and the return of acid secretion following such inhibition requires de novo synthesis of new pump. Although these drugs have brought about remarkable changes in ulcer therapy, the efficiency of these drugs is still debatable. Reports on clinical evaluation of these drugs shows that there are incidences of relapses and adverse effects and danger of drug interactions during ulcer therapy. Further, in the developing countries, like India, most of the population is living in rural areas and depending on their indigenous system of drugs because of expensive modern treatment. Hence, the search for an ideal anti-ulcer drug continues and has also been extended to herbal drugs in search for new and novel molecules, which afford better protection, decrease the incidence of relapse and decrease the cost of the treatment. Of late the search for new safe alternative drugs have rekindled the interest in cytoprotective drugs, which protects the mucosal layer from inducing agents. Cytoprotection has been defined as the ability of pharmacological agents-originally prostaglandins to prevent or reduce gastric, duodenal, or intestinal mucosal injury by mechanisms other than inhibition of gastric acid secretion. Although few drugs like sucralfate and prostaglandin analogs are recognized as cytoprotective agents (Vergin and Kori-Lindner, 1990), many natural drugs have been reported to posses this activity viz. plantain banana (Musa sapientum var Paradisiaca), Tectona grandis, Azadirachta indica and rasayana drugs like Centella asiatica, Asparagus racemosus, Convolvulus pluricaulis, Emblica officinalis, Bacopa monniera and Withania somnifera, etc. (Goel and Sairam , 2002). India is one of the country rich in medicinal plants which were used by our ancestors. Traditionally, plants were used as medicine in a traditional way such as Ayurveda, Naturopathy, Siddha and Unani. After knowing the used of plants in medicine, synthetic drugs were now started replacing by herbal products. Humans have used plant materials since prehistoric times and in some countries such as China documentary evidence shows that herbal medicines have been used for at least 7000 years. In Europe there is a rich history in the use of herbal medicines and these have been well documented in medieval herbals such as Culpepers and Gerards materia medica. Nowadays many countries are having interest in using Indian medicinal plants as it can cure many diseases and other purposes. Herbal medicines differ from synthetic drugs in several attributes. Though, herbal medicines are mixed chemical compounds, all have not been isolated, characterized and quantified. When an extract of a plant or a compound isolated from the plant has to be clinically evaluated for a therapeutic effect not originally described in the texts of traditional systems or, the method of preparation is different, it has to be treated as a new substances or new chemical entity. The same type of acute, subacute and chronic toxicity data has to be generated. Majority of medicinal herbs contain dozens of different compounds, often of great complexity, flavonoids, saponins, glycosides, alkaloids, mucilage, tannins, polysaccharides, etc., that buffer, modulates and modify the effect of any active principles. Study after study has shown that administering of isolated purified constituents of the whole or part of the plant cannot mimic effects produced by extracts of same part of the plant. 1.3.1 Pathophysiology of Peptic Ulcer Peptic ulcer generally occurs when aggravating factors are higher than defensive mucosal factors(Goel Bhattacharya, 1991). The peptic ulcer can be treated by reducing the activity of aggravating factors thereby increasing the activity of defensive factors. H. Pylori H.Pyloric is a Gram -ve spiral-shaped bacterium. H.Pyloric is the most common cause of non-NASID associated peptic ulcer disease. H.Pyloric has been found in the gastric antrum of a significant number of patients with duodenal ulcers and gastric ulcers. H.Pyloric lives in the acidic environment of the stomach. The initial infection is transmitted by the oral route. H.Pyloric attaches to adhesion molecules on the surface of gastric epithelial cells. In the duodenum, H.Pyloric attaches only to areas containing gastric epithelial cell that have arisen as a result of excess and damage to the duodenal mucosa. H.Pyloric is able to live in such a hostile environment partly because of its production of the enzyme increase, which converts urea to ammonia. The ammonia buffers the H+ and forms ammonia OH creating an alkaline cloud around the bacteria and protecting it from the acidic environment of the stomach. It causes inflammation and epithelial cell damage (Golan, and Arman). Nsaids More than 100, 000 patients are hospitalised each year for NSAID-associated gastro-intestinal complications and gastrointestinal bleeding has a 5% to 10% mortality rate in these patients. NSAID-associated gastrointestinal damage is attributable to both topical injury and systemise effects of the NSAID. Most NSAIDS are weak organic acids. In the acidic environment of the stomach, these drugs are neutral components that can cross the plasma membrane and enter gastric epithelial cells. In the neutral intracellular environment, the drugs are recognized and trapped. The resulting intracellular damage is responsible for the local gastrointestinal injury associated with NSAID use. Gastric acid secretion Inhibition of cycloxygenase prostaglandins Bicarbonate/ Mucous Production Blood flow NSAIDS Neutrophil adherence Mucosal dama- Expression of intercellular vascular endothelial ge due to neutro- Adhesion molecules in gastric cells phil free radicals Vascular endothelium and proteases. Acid hypersecretion Acid hypersection is an important causative factor in some patients with peptic ulcer disease. Zollinger-Ellison Syndrome(Z-E-S)are two clinical examples in which hyperacidity leads to peptic ulcer disease. In Z-E-S, a gastrin-recreting tumor of the non-beta cells of the endrocrine pancrease lead to increased acid secretion. In lusting ulcer, seen in patients with reverse head injuries, heightened vagaltone causes gastric hyperacidity. Figure is showing the interactions among an enterochromaffin-like (ECL) cell that secretes histamine, a parietal cell that secretes acid, and a superficial epithelial cell that secretes cytoprotective mucus and bicarbonate. Physiological pathways, shown in solid black, may be stimulatory (+) or inhibitory (-). 1 and 3 indicate possible inputs from postganglionic cholinergic fibres, while 2 shows neural input from the vagus nerve. Physiological agonists and their respective membrane receptors include acetylcholine (ACh), muscarinic (M), and nicotinic (N) receptors; gastrin, cholecystokinin receptor 2 (CCK2); histamine (HIST), H2 receptor; and prostaglandin E2 (PGE2), EP3 receptor. Drug actions are indicated by dashed lines. A blue X indicates targets of pharmacological antagonism. A light blue dashed arrow indicates a drug action that mimics or enhances a physiological pathway. Shown in blue are drugs used to treat acid-peptic disorders. NSAIDs are nonsteroidal anti-inflammatory drugs and are ulcerogenic. 1.4 Gastrointestinal Motility: Laxatives are drugs that either accelerate faecal passage or decrease faecal consistency. They work by promoting one or more of the mechanisms that cause diarrhoea. Because of the wide availability and marketing of OTC laxatives, there is a potential that an appropriate diagnosis will not be sought (Jahangir moini). Rarely in medicine is there an absolute indication for the use of laxatives. A high fibre, well-balanced diet rich in fruits and vegetables supplemented by bran should be enough to normalize bowel function. The fear of autointoxication and the constant concern of many patients regarding the frequency and quality of bowel movement make laxatives one of the most popular over the counter drugs in the market with serious potential for user abuse. Accepted indications for laxatives and stool softeners include preparation for diagnostic colonic examination (Barium enema, colonoscopy: treatment of anorectal disorders) and prevention of hepatic encephalopathy (Smith and Reynard). Fibre is defined as the undigested residue of fruits, vegetables, and other foods of plant origin after digestion by the human GI enzymes. Fibres water holding capacity is the ability of the fibre to hold water and make bulking of faecal materials possible. Fibres stool bulking capacity is the ability of the fibre to increase the volume of intestinal content because it can absorb and holds water. Bacterial growth in the colon provides additional bulking. Insoluble fibres speeds GI transit time. Cholinergic mechanisms are also responsible for modulating motor phenomena in the gut; thus it is not surprising that cholinomimetic agents are effective in promoting gastrointestinal motility. It also has cholinomimetic properties, apparently sensitizing intestinal smooth muscle cells to the action of Acetylcholine rather than acting on acetylcholine receptors. The drugs acts to hasten esophageal clearance, raise lower esophageal sphincter pressure, accelerate gastric emptying, and shorten small bowel transit time 1.4.1 Mechanism of Laxative Action: (KD Tripathi, 1999) a. By their hydrophilic or osmotic nature, laxative can cause retention of fluids, in the colonic content, as well as increase the mass. b. Inhibiting Na+K+ATPase of villous cells impairing electrolyte and water absorption. c. Stimulating adenyl cyclase in crypt cells increasing water and electrolyte secretion. d. Enhancing PG synthesis in mucosa which increases secretion. e. Structural injury to the absorbing intestinal mucosal cells. 1.4.2 Classification of Laxatives:- Many drugs in low doses act as laxative and in larger doses as purgatives 1. Bulk forming 4. Stimulant purgatives Dietary fibre: Bran (a) Diphenylmethanes Psyllium Phenolphthalein Methylcellulose (b) Castor oil 5. Anthraquinones 2. Stool softner Senna, Cascara sagrada Docusates (DOSS) 6. Osmotic purgatives 3. Lubricant Magnesium, sodium salts Liquid paraffin Lactulose 1.4.3 Anthraquinone Derivatives:- Senna is obtained from the leaves and pods of Cassia augustifolia and contains the anthraquinone glycosides called emodins. In oral dose the sennosides is poorly absorbed, but after removal of the sugar and reduction to anthrol by colonic bacteria, they are absorbed into circulation excreted in bile to act on small intestine. It takes 6-7 hrs to produce action. The active principle is believed to act on the myentric plexus to increase peristalsis and decrease segmentation. They also inhibit salt and water absorption in the colon. In India, sennosides are usually marketed in combination with stool softeners such as docusates. Side effects observed are nausea, vomiting, diarrhoea, colic, urine discoloration (yellowish brown to red) and melanosis (colonic atony and mucosal pigmentation after a regular use of the drug). It should be used cautiously in women and children bebelow years of age, and after abdominal surgery. Preparations GLAXENNA tab. Sennosides ( calcium salt) 11.5 mg. SOFSENA tab 12 mg. PURSENNID tab -18 mg LAXSENA tab 12mg, 18mg(forte)

Analysis of The Lost World by Michael Crichton Essay -- The Lost World

Analysis of The Lost World by Michael Crichton Michael Crichton's novel, The Lost World began with the exposition of a character who is infamous to Crichton's work, Ian Malcom. The entire introduction and prologue is about Malcom and his scientific views and theories. In a section of the book called 'Hypothesis';, Malcom discusses a theory of 'lost worlds'; - areas in which extinct beings may live, with Richard Levine, a man who's ideas were totally different from Malcoms. Levine and Malcom discuss a possible journey to an island that is suspected to be one of the so-called 'lost worlds';. At first, Malcom is reluctant and thinks that the idea is stupid. After much coaxing, Malcom agrees to go. Levine arrives on the island first, after a lot of useless chatter and talk and the introduction of two young characters, Arby and Kelley, who are students of Levine's and sometimes run errands for him. Almost immediately after they arrive, Levine and his assistant, Diego, begin to search for clues to what the science community calls 'aberrant forms';, which many people have spotted but have been unable to identify. They come to a stream bed, where they are attacked by a group of unidentifiable animals. They capture and kill Diego, and Levine is nearly killed. Malcom and his team of field researchers finally make it to the island - and Arby and Kelley stow away in one of the many high-tech trailers that they had taken on their expedition. After the first day or so goes smoothly, chaos breaks out. When three men, Dodgson, King, and Basselton decide to go to the island, thinking that no one has been there. They plan on stealing the dinosaur eggs and breeding them. In doing so, they cause a ruckus on the island, and the dinosaurs begin to lose their patience. They go on a killing spree, which is graphically described by Critchton as well as nearly killing Arby. The climax of the story comes when Malcom and Sarah Harding (one of his associates) get trapped in a trailer that is about to tumble off the ledge of a cliff. Luckily for them, they are saved by another of their colleagues, named Richard Thorn, saves them by using Dodgson's jeep (which they had thought was broken down, AND after they thought all three of the egg thieves were dead) and ties a rope to the trailer and miraculously saves them. The story isn't over yet, though. The group mi... ... putting together trailers for their expedition and they tell the boss that they are too weak and something could happen, but the boss ignores thim. It's so obvious, but it is foreshadowing. Also, another classic example of foreshadowing from the Jurassic Park series is the shaking and rumbling of the ground then a calm for a second or two before the t-rex attacks. Once again, obvious, but it's still an example of foreshadowing. All in all, this book was good even though I do not enjoy reading. Despite many things that I thought could have been clarified and in a lot of instances, were so dumb that they insulted me, I was entertained by the book. It had a little bit of everything†¦Mind games, action, gore and death, and what I thought was the best part of the entire book†¦Great characters. Between the most important characters, such as Malcom, to the lower-status ones such as Sarah Harding, or even Kelley and Arby's parents, who never actually appear†¦All of them were greatly put together. I might not be able to recommend this book to you if you are reading for pleasure, but if there was one book that you absolutely had to read, Michael Crichton's The Lost World has to be it.

A Poetry Explication :: Shakespeare Sonnet Poem Essays

A Poetry Explication "Poetry is the revelation of a feeling that the poet believes to be interior and personal [but] which the reader recognizes as his own." (Salvatore Quasimodo). There is something about the human spirit that causes us to rejoice in shared experience. We can connect on a deep level with our fellow man when we believe that somehow someone else understands us as they relate their own joys and hardships; and perhaps nowhere better is this relationship expressed than in that of the poet and his reader. For the current assignment I had the privilege (and challenge) of writing an imitation of William Shakespeare’s "Sonnet 87". This poem touched a place in my heart because I have actually given this sonnet to someone before as it then communicated my thoughts and feelings far better than I could. For this reason, Sonnet 87 was an easy choice for this project, although not quite so easy an undertaking as I endeavored to match Shakespeare’s structure and bring out his themes throu gh similar word choice. As far as structure goes, Shakespeare’s sonnet and my own are very similar. My 14-line sonnet is written in Iambic Pentameter and follows the abab-cdcd-efef-gg rhyme scheme within three quatrains and a couplet, as does that of Shakespeare. For this reason, my poem follows a meter very close to Sonnet 87. This is also due to the fact that both sonnets contain 10 syllables per line, as well as an eleventh in most lines that derives from weak, or "feminine" verb endings such as "keeping" in the first line of my poem, and "possessing" in that of Shakespeare. Thus, the finality residing in the 10 syllable lines that use masculine endings is accounted for. In addition, my sonnet mirrors Sonnet 87 by starting with "farewell" and ending with Shakespeare’s couplet modified, and one can see his influence in the third quatrain of my poem, which echoes back the last 4 lines of Sonnet 87’s third quatrain. In this way, I was able to mimic most of Shakespeare’s structure while inserting phrases of my own. Throughout the poem I attempted to remain true to Shakespeare’s sonnet by way of word choice, while adding my own twist. My poem alternates between the more archaic (but arguably more beautiful) "thou" and the more modern "you". This is done to tie my poem, written in the present, to Shakespeare’s work of the past.

Paper Towns Book Response Essay

The? Book? Talk? visual? which? caught? my? eye? the? most? was? Clare? Considine’s? project? on? ‘Paper? Towns’? by? John? Greene.? After? talking? with? her? I? learned? the? book? is? about? two? young? people,? Margo? and? Quentin? who? navigate? through? their? senior? year? in? the? suburbs? of? Orlando,? Florida? in? the? early? 2000’s.? When? exposed? to? a? dead? man? who? has? committed? suicide? in? Jefferson? park,? Margo,? at? 10? years? old? says? â€Å"Maybe? all? the? strings? inside? him? just? broke. †? After? this? moment,? the? two? continually? link? human? emotions? to? acting? as? â€Å"strings†? throughout?the? novel.? In? the? middle? of? the? book? Margo? runs? away? and? the? plot? is? transformed? to? Quentin’s? finding? her? on? a? long? road? trip? with? his? two? best? friends? Ben? and? Radar? and? Ben’s? girlfriend? Lacey.? The? two? create? idealist? views? of? each? other? which? only? by? the? end? is? proven? wrong? by? reality.? In? the? visual,? there? is? a? series? of? 6? maps? showing? the? states? which? are? driven? through? in? the? novel? to? find? Margo,? North? Carolina,? South? Carolina,? Pennsylvania,? New? York,? Virginia,? and? Florida.? Starting? at? the? front? of? the? map? and? ending? at? the? end,? there? is? a? path? which? takes? the? viewers? of? this? visual? through? the? plot? of? the? story.? Also,? there? are? various? quotes? spread? about? the? map? to? support? the? explanation? of? the? plot.? To? begin,? the? trail? starts? at? two? houses,? Margo? and? Quentin’s.? Starting? here,? the? black? tacks? represent? Margo? and? the? white? tacks? show? Quentin.? This? contrast? to? me? exemplifies? a? ying? yang? situation.? Next,? the? string? goes? down? to? four? houses:? Lacey,? Jace,? Becca,? and? Karen’s,? whose? houses? have? a? spraypainted? ‘M’? on? them.? This? ‘M’? symbolizes? Margo’s?  anger? toward? them? because? her? ex? boyfriend? Jace? cheated? on? her? with? Becca? her? best? friend,? and? this? was? kept? a? secret? by? Karen? and? Lacey? her? are? her? close? friends? also.? It? then? leads? down? to? Sea? World? which? they? break? into? and? then? the? path? leads? to? the? sun? tower.? At? the? sun? tower? Margo? says? the? significant? quote? â€Å"All? those? paper? people? living? in? their? paper? houses,? burning? the? future? to? stay? warm.? All? the? paper? kids? drinking? beer? some? bum? bought? for? them? at? the? paper? convenience? store.? Everyone? demented? with? the? mania? of?  owning? things.? All? the? things? paper? thin? and? paper? frail.? All? all? the? people,? too.? I’ve? lived? here? for? eighteen? years? and? I? have? never? once? in? my? life? come? across? anyone? who? cares? about? anything? that? matters. †? After,? the? track? leads? to? two? paper? people? with? Margo? and? Quentin’s? tacks? on? them? suggesting? they? are? too? one? dimensional? paper? people? by? this? point.? Following,? Margo’s? tack? then? moves? into? a? question? mark? when? she? runs? away.? By? this? point? all? Quentin? knows? is? she? is? going? to? a? ‘Paper? Town’.? Quentin’s? tacks? continue? into?  subdivisions? in? Orlando,? Florida? where? he? begins? his? search? with? thoughts? that? by? ‘Paper? Towns’? she? is? referring? to? the? bland? subdivisions? with? nothing? ever? changing? but? does? not? find? her? there.? The? next? tack? is? at? a? barn? where? he? sees? Margo? has? spraypainted? the? words,? â€Å"You? will? go? to? Paper? Towns? and? you? will? never? return. †? There? he? realizes? Paper? Towns? are,? by? literal? definition,? fake? towns? choreographers? put? on? their? maps? to? prevent? plagiarism.? This? leads? him? to? Algoe,? New? York,? (represented? by? the? car? on? the? visual)? where? he?  finds? Margo? in? a? barn.? The? last? part? to? the? visual? is? a? little? booklet? with? ‘expectations,? assumptions? and? idealism’? written? on? the? pages,? illustrating? the? journal? Margo? keeps,? writing? down? all? to? happen? to? her? including? her? idealizations? for? Quentin.? This? points? out? the? moment? when? Quentin? and? Margo? realize? they? had? been? idealizing? one? another? to? be? something? they? are? not? the? whole? time? and? so? they? decide? to? part? ways.? Ultimately? this? visual? is? very? thorough? and? creative? and? illustrates? the? theme? of? the? ideal? person? being? nonexistent.

Rank of Icici Stock Minds

Sheet1 RANK 1 2 3 4 PARTICIPANT NAME COLLEGE NITIN GAJANAN MALUSARE JANARDAN INSTITUTE GIRISH MAHESHKUMAR SAVLANI K S BUSINESS SCHOOL PRATEEK GOVILA BIT MESRA PUNE UNIVERSITY DEPARTMENT OF KISHAN BHATTAD MANAGEMENT SCIENCES (PUMBA) CITY MUMBAI AHMEDABAD RANCHI PUNE PORTFOLIO 1666999. 9 1666553. 6 1666432. 5 1662356. 05 TURNOVER 16935103. 38 19998020 19946293. 4 0 % GAIN 11. 13% 5. 67% 9. 36% 0. 00% Basis highest % change in profit compared to the previous day portfolio COMMENTS REMARKS 6 7 8 9 10 11 12 13 14 15 16 JIGAR B SHAH SHANTI BUSINESS SCHOOL INDIAN INSTITUTE OF MANAGEMENT SAGAR CHOUDHURY (IIM) ISHAN ONKAR TRIVEDI NIRMA INSTITUTE OF MANAGEMENT ANIL KUMAR YADAV IMS GHAZIABAD RAVINDER REDDY IIT – MADRAS (MGMT DEPT) JAY PRAKASH MEHTA KANDIVALI EDUCATION SOCIETYÂ   INDIAN INSTITIUTE OF PARV DHIMAN MANAGEMENT(IIM) LALA LAJPATRAI INSTITUTE OF DHIRAJ KUNDANMAL SONI MANAGEMENT KALPESH RAJPUT B. K.INSTITUTE OF MANAGEMENT CHETANA'S INSTITUTE OF ROHAN PRAVIN DHUNDUR MANAGEMENT AN D RESEARCH PRIYANKA BHIKHABHAI CHAUDHARY INSTITUTE OF PATEL TECHNOLOGY VISHWAKARMA INSTITUTE OF DINOOP HARIDAS NAIR MANAGEMENT PARLE TILAK VIDYALAYA ASSOCIATION INSTITUTE OF VIJAY DALCHAND SOLANKI MANAGEMENT(PTVAIMS) AHMEDABAD ROHTAK AHMEDABAD DELHI CHENNAI MUMBAI KOLKATA MUMBAI AHMEDABAD MUMBAI GANDHINAGAR PUNE 1633101. 34 1630987. 35 1624240. 56 1607499. 08 1603541. 45 1592069. 45 1587710. 2 1583445. 77 1577859. 05 1571068. 17 1569750 1569228. 22 0877028. 01 19601921. 9 18727524. 25 19529775. 94 13647528. 25 17734802. 6 17597289. 8 13905246. 26 8914119. 7 10081802. 57 880898 18632832. 41 11. 75% Mobile Winner – 14. 03. 13 4. 13% 8. 37% -2. 13% 3. 39% 2. 62% 4. 80% 5. 41% 3. 26% 4. 74% 4. 72% 5. 41% 17 18 19 20 21 22 23 24 25 26 27 28 29 MUMBAI 1568526. 09 1558883. 62 1551591. 65 1551477. 36 1548478. 45 1544469. 5 1542588. 9 1540635. 82 1538577. 6 1536670. 52 1536123. 59 1533144. 14 1531809. 2 9199916 19665170. 27 19578787. 6 9171863. 97 10972784 16540805. 5 3318021. 6955868 . 33 3697253. 9 10295002. 38 18981106. 07 17478175. 1 13408401. 9 5. 97% 2. 78% 2. 33% 3. 31% 1. 95% 2. 96% 3. 11% 3. 28% 4. 15% 2. 62% 1. 97% 1. 98% 2. 12% JAY VITTHALBHAI SANGANI MARVADI INSTITUTE OF MANAGEMENT RAJKOT SOM LALIT INSTITUTE OF KUSHAL AMIT PARIKH AHMEDABAD MANAGEMENT BIJAL BHARATH SELARKA SRM SCHOOL OF MANAGEMENT CHENNAI DHAVAL RAJENDRA DESAI MUMBAI EDUCATIONAL TRUST (MET) MUMBAI PARIN MARU MUMBAI KJ SOMAYA KETAN POPATBHAI S K PATEL INSTITUTE OF DHAMELIYA MANAGEMENT GANDHINAGAR RONAK JAYSUKH SODHA N.L. DALMIYA COLLEGE MUMBAI VISHWA VISHWANI SCHOOL OF PREM KUMAR HYDERABAD BUSINESS PRARTHANA CHANDAR UNITED WORLD SCHOOL OF AMIN BUSINESS MUMBAI SYMBIOSIS COLLEGE OF ARTS & GEORGE PARAPUZHA JOB COMMERCE PUNE LALSINGH SANGRAMSINGH S. B PATIL INSTITUTE OF RAJPUT PUNE MANAGEMENT SUDEEPTA SARMA IILM INSTITUTE FOR HIGHER BORUAH DELHI EDUCATION (IILM ) INSTITUTE OF MANAGEMENT DEVELOPMENT AND RESEARCH (IMDR) AGNEL CHARITIES AGNEL SEVA SANGHA'S FR. C. RODRIGUES INST. OF MANAGEMENT. STUDIES RUSTOMEJEE BUSINESS SCHOOL DAYANANDA SAGAR ACEDEMY OF TECHNOLOGY AND MANAGEMENT MUMBAI INSTITUTE OF MANAGEMENT & RESEARCH GURU NANAK INSTITUTE OF MANAGEMENT STUDIES (GNIMS) INDIAN INSTITUTE OF KNOWLEDGE MANAGEMENT (IIKM) AMITY GLOBAL BUSINESS SCHOOL ACCURATE BUSINESS SCHOOL ATHARVA SCHOOL OF BUSINESS NSTITUTEÂ   OF TECHNOLOGY AND MANAGEMENT- INSTITUTE OF FINANCIAL MARKETS (ITM-IFM) RAWAL INSTITUTE OF ENGINEERING & MANAGEMENT KIRLOSKAR INSTITUTE OF ADVANCED MANAGEMENT STUDIES CITY COLLEGE INDIAN INSTITUTE OF TECHNOLOGY PARUL INSTITUTE OF MANAGEMENT INDIAN INSTITIUT OF MANAGEMENT(IIM) MEENAKSHI SUNDARAJAN SCHOOL OF MGMT JAIPURIA INSTITUTE OF MANAGEMENT STUDIES H. K.INSTITUTE OF MANAGEMENT, STUDIES& RESEARCH, JOGESHWARI CHRIST COLLEGE 30 DIVYA RANI SINGH PUNE 1530024. 8 1504035. 81 1. 22% 31 32 33 34 35 36 37 38 39 NIKHITA VINAY RANE BHAUMIK KIRTIKUMAR MEHTA SANTOSH KUMAR ANAND PRAKASH YADAV HARMEET HARBINDER ALAG PRAVEEN GANESAN SASANK BANDARUPALLI MD MOHSIN KHAN SNEHAL SHIVA JI SHINDE MUMBAI MUMBAI BANGALORE MUMBAI MUMBAI CHENNAI HYDERABAD DELHI MUMBAI 1529786. 95 1528691. 21 1526535. 5 1526029. 7 1525450 1523362. 3 1522990. 93 1522771. 22 1522282. 7 1514444. 46 0 948645. 9 1174189. 5 0 19633466. 4 7175048. 23 15763374. 15 11326878. 5 1. 99% 0. 00% 1. 83% 2. 37% 2. 29% 1. 56% 1. 53% 0. 94% 0. 54% 40 41 42 43 44 45 46 47 48 49 50 51 52 53 54 55 56 57SAURAV SHARMA PARVEEN GARG ARPAN KUMAR DAS MADHU SUDAN MC SURENDRA BABU GADWALA DIPAK RASIKBHAI KATHROTIYA SHIVANK GOEL DEVIKA RAJU ASHISH RANA BHAVESH HARISH AGARWAL SASWATA GHOSH RAVIPRAKASH RAMSNEHI PRASAD MILAN A THAKKAR NAVEEN KUMAR BHATNAGAR GOVIND PRATAP SINGH MERTIYA BONY KURIAN PUNNOOSE ANSHUL BANSAL ABHISHEK GOYAL SHAIKH MOHD RAMEEZ MUKHTAR KUSHALAPPA B K JABEZ E DANIEL HARDIC VORA VAISAKH E J MUMBAI DELHI PUNE BANGALORE KANPUR BARODA SHILLONG CHENNAI DELHI MUMBAI BANGALORE 1520505. 07 1517381. 9 1516829. 38 1516420. 05 1516038. 59 1515656. 75 1515339. 65 1515279. 58 1515096. 5 1513370. 8 1513242 15 13070. 33 1512952. 06 1512904. 4 1512802. 4 1512655. 28 1512567. 22 1512270 1635277. 67 2360833. 43 3211287. 58 2450275. 45 2141875. 73 17414304. 45 0 2676943. 84 0 13356288. 94 99393 6559817. 75 18790067. 43 0 0 9150470. 72 17570914 0 1. 77% 0. 97% 1. 12% 1. 09% 1. 15% 0. 57% 0. 34% 0. 95% 1. 94% 0. 64% 0. 76% 0. 91% -8. 71% 2. 66% 1. 22% 1. 67% 1. 03% 2. 11%IIPM AHMEDABAD PUNE AMITY GLOBAL BUSINESS SCHOOL INSTITUTE OF MANAGEMENT STUDIES DEHRADUN (IMS) JK BUSINESS SCHOOL AMITY GLOBAL BUSINESS SCHOOL IIMB MAHARISHI ARVIND INSTITUTE OF SCIENCE AND MANAGEMENT JSPM'S ABACUS INSTITUTE OF COMPUTER SCIENCE AND MANAGEMENT BNMIT BISHOP HEBER COLLEGE IEIBS AKADEMIA REGIONAL INSTITUTE OF COOPERATIVE MANAGEMENT MAHARASHTRA INSTITUTE OF TECHNOLOGIES-SCHOOL OF TELECOM (MIT SOT) GLOBSYN TECHNOLOGIES LTD JAYWANTRAO SAWANT INSTITUTE OF MANAGEMENT RESEARCH SKYLINE BUSINESS SCHOOL CAMP EDUCATION SOCIETY (INSTITUTE OF MANAGEMENT) ATMIYA INSTITUTE OF MANAGEMENT INDIAN INSTITUTE OF PLANNING & MANAGMENT (IIPM, CHATTARPUR) INTERNATIONAL SCHOOL OF MANAGEMENT EXCELLENCE DELHI CHENNAI BANGALORE JAIPUR 58 59 60 61 62 PUNE BANGALORE TRICHY MUMBAI BANGALORE 1512104. 65 1511085 1509935 1509755. 86 1509406. 55 0 0 0 2709639. 3 0 1. 10% 0. 91% 0. 99% 2. 31% 1. 00% 63 64 65 66 67 68 69 70CHANCHAL KANSAL SUBHASIS SAHA PUNAM ANNASO BANDAL SONU SINGHAL RAJESH MAHADEV ANDIRAO PARESH ISHVAR BHAI MITHANI SAMRAT THATIKONDA TWARIT S TARPARA PUNE KOLKATA PUNE DELHI PUNE RAJKOT DELHI BANGALORE 1509405. 94 1509016. 5 1508847 1508597. 32 1508523 1507986. 75 1507803. 6 1507612 1000214. 4 0 0 4107663. 24 0 0 2278392. 99 119175 1. 35% 1. 33% 0. 71% 0. 57% 1. 07% 0. 43% 0. 93% 0. 85% Page 1 Sheet1 71 72 73 74 75 76 POOJA BALU VETAL RAJESH KUMAR PARAG M LATHIYA ASHUTOSH SINGH ARJUN M MAYANK SINHA ST. MIRA'S COLLEGE FOR GIRLS SCHOOL OF COMMUNICATION & MANAGEMENT STUDIES (SCMS) LDRP BENGAL INSTITUTE OF BUSINESS STUDIES(BIBS) ST. JOSEPH'S INSTITUTE OF MANAGEMENT AMITY UNIVERSITY B. P.PODDAR INSTITUTE OF MANAGEME NT AND TECHNOLOGY (BPPIMT) CMS BUSINESS SCHOOL INDIRA INSTITUTE OF BUSINESS MANAGEMENT FUTURE BUSINESS SCHOOL GREAT LAKES INSTITUTE OF MGMT PUNE KOCHI GANDHINAGAR KOLKATA TRICHY GURGAON 1507374. 25 1507298. 7 1507284. 66 1506689. 12 1506507 1506000 632126. 6 811641 4166933. 75 137269. 44 1051965 334500 0. 49% 0. 49% 0. 78% 0. 65% 0. 49% 0. 52% 77 78 79 80 81 82 83 84 85 86 87 88 89 90 91 92 93 94 95 96 97 98 99 100 101 102 103 104 105 SANJANA SEN RAHUL NICHANAMETTLA PRATHAMESH DHARMIK MANDALE POOJA SETHI SANTHOSH KUMAR S SUDHANSHU KESHAV MOURYA ANURAG GUPTA RAM MILAN KANNUJIA KOLKATA BANGALORE MUMBAI KOLKATA CHENNAI MUMBAI JALANDHAR DELHI KOLKATA PUNE KOLHAPUR AHMEDABAD KOLKATA DELHI PUNE JALANDHAR TRICHY AHMEDABAD VISNAGAR JAIPUR 1505896. 5 1505846. 1505772 1505472. 76 1505448. 66 1505371 1505182. 5 1505154. 7 1504923. 27 1504847 1504450. 94 1504090. 2 1503889. 46 1502875 1502740. 5 1502332 1502277 1502240. 76 1501853. 5 1501507. 86 1501469. 41 1501321 1501252. 75 1501160 1501127. 4 1501077. 05 1500834. 35 1500709. 1 1500515 0 510517. 1 0 243199 16911647. 35 0 0 0 0 1478127. 6 0 16151721. 7 498939. 5 0 714160 450073. 2 0 17700875. 4 0 448070. 76 1047920. 95 0 51487. 28 418670 1032958. 65 0 0 415179. 6 0 0. 33% 0. 33% 0. 61% 0. 57% 0. 23% 0. 52% 1. 33% 0. 60% 0. 54% 0. 12% 0. 00% 0. 87% 0. 81% 0. 48% 0. 15% 0. 08% 0. 13% 1. 41% 0. 64% 0. 10% 1. 34% 0. 12% 0. 12% 0. 08% -0. 08% 0. 38% 0. 03% 0. 08% 0. 03%SHRI VISHVESARIYA IMS LOVELY PROFESSIONAL UNIVERSITY GAUTAM BUDDHA UNIVERSITY INTERNATIONAL INSTITUTE OF SUKANTA SAHA BUSINESS STUDIES (IIBS) SADHU VASVANI INSTITUTE OF BHUMIKA NIRMAL SONI MANAGEMENT (SVIMS) SANJAY GHODAWAT INSTITUTE SOURABH INANI (SEMINAR II) VARSHIT BIPINBHAI DOSHI NRIMB AMINUR RAHAMAN GAZI NSHM BUSINESS SCHOOL(NBS) ASIA PACIFIC INSTITUTE OF RENU YADAV MANAGEMENT NASIRUDDIN FAKRUDDIN DR. D. Y. PATIL INSTITUTE OF SHAIKH ENGINEERING & TECHNOLOGY APEEJAY INSTITUTE OF ANU THANGRIA MANAGEMENT TECHNICAL CAMPUS MAREDDY INDIAN INSTITIUT OF MARKANDEYU LU MANAGEMENT(IIM) INDIAN INSTITIUT OF SIDDHARTH M MANAGEMENT(IIM) NARESH DAHYABHAI VISNAGAR INSTITUTE OF PRAJAPATI MANAGEMENT MEGHA AGARWAL BANASTHALI VIDYAPITH CAMPUSJAY PRAKASH CHAUDHARY OXFORD COLLEGE OF MANAGEMENT BANGALORE LOKESH DURGESH GHOSAL ITM DOMBIVLI MUMBAI SRN ADARSH COLLEGE OF ASHISH KUMAR MISHRA BANGALORE MANAGEMENT ASHWINI DEVARAJAM SANKA MUMBAI VIVA COLLEGE OF MANAGEMENT INTERNATIONAL MANAGEMENT AMANDEEP SINGH MAINI DELHI INSTITUTE(IMI) RAJ KUMAR GOYAL INSTITUTE FOR ABHISHEK RAI DELHI TECHNOLOGY (RKGIT) VISHAL JALHOTRA DEHRADUN GRAPHIC ERAÂ   UNIVERSITY MONICA SATISH SHARMA ICL COMMERCE COLLEGE VASHI MUMBAI NANDALAL M SIVADAS AMRITA COLLEGE OF MANAGEMENT BANGALORE BABU BANARASI DAS NATIONAL INSTITUTE OF TECHNOLOGY & MANAGEMENT VIDYALANKAR INSTITUTE OF SUSHMA VIJAY GHADGE MANAGEMENT LALIT SONI ST.KABIR INSTITUTE MANAGEMENT NITU SINHA MERI INSTITUTE MONICA ALLURI NSB KENGUA ASHUTOSH ICFAI BUSINESS SCHOO L(IBS) DILIP MADHUBHAI CENTRE FOR MANAGEMENT STUDIES PRAJAPATI (CMS) SUJITH S KURUP RAI BUSINESS SCHOOL INSTITUTE OF BUSINESS PIYUSH DILIP KHESE MANAGEMENT & RESEARCH (IBMR) TUSHAR GULATI ICFAI BUSINESS SCHOO L(IBS) JAYDEEP PRATAPRAI INDIAN EDUCATION SOCIETY RATHOD INSTITUTE OF MANAGEMENT(IESIMS) INTERNATIONAL SCHOOL OF AVIJIT SAHA BUSINESS JAGANATH SUBRAMANIAN HYDERABAD BUSINESS SCHOOL SOUMYA SUKHMAY NATIONAL INSTITUTE OF CHATTERJEE COOPERATIVE MANAGEMENT (NICM) KALPESH KAILASH SHARMA MDI PRIYANT RAMESHBHAI LADANI OXFORD COLLEGE OF MANAGEMENT YASH YOGESHBHAI SHAH K S BUSINESS SCHOOL 2 DR. GAUR HARI SINGHANIA INSTITUTE PUSHPINDER SINGH PURI OF MANAGEMENT & RESEARCH INDIAN INSTITUTE OF MANAGEMENT SASIKANTH BAPATLA (IIM) ABHISHEK MADASSERY FEDERAL INSTITUTE OF SCIENCE & ASHOK TECHNOLOGY(FISAT) RIZVI INSTITUTE OF MANAGEMENT KARAN KIRTI DOSHI STUDIES R. A.PODAR INSTITUTE OF AKSHIT BAJ MANAGEMENT ACHARYA'S BANGALORE BUSINESS SURI SAI SRIKANTH SCHOOL INDIAN INSTITUTE OF PLANNING AND VIREN PARMAR MANAGEMENT(IIPM) SREE NARAYAN GURUKULAM SHAHANAS PA COL LEGE OF ENGINEERING FABEESH MOHAMED IIPM, KOCHI G SANDEEP GANDHIKOTA BUSINESS SCHOOL DEEPAK HEDDA GARDEN CITY COLLEGE KOLHAPUR INSTITUTE OF VENUGOPAL LALIT DAVDA TECHNOLOGY INSTITUTE OF BUSINESS AND SAISMITA DALAI COMPUTER STUDIES(IBCS ) SOUMITRA KUMAR INDIAN INSTITUTE OF PLANNING & MANDAL MANAGEMENT(IIPM) DIPTI DINESH NADKARNI IEIBS AKADEMIA BLESSINA JEBARAJ JEPPIAR ENGINEER COLLEGE SYMBIOSIS INSTITUTE OF BUSINESS STUTI BANSAL MANAGEMENT (SIBM) INDIAN INSTITUTE OF PLANNING & P SUNIL KUMAR MANAGEMENT(IIPM) JAYAMUKHI INSTITUTE OF GOUTHAM BIJJALA TECHNOLOGICAL SCIENCES DEBASISH BAJ ITM BUSINESS SCHOOL ORIENTAL INSTITUTE OF ANIRUDH MAHESH SANGHI MANAGEMENT VIVEK AGARWALA LOVELY PROFESSIONAL UNIVERSITY VELS INSTITUTE OF BUSINESS DASARATHY GANESAN ADMINISTRATION GANESH R NIT DEEPMALA YADAV 106 107 108 109 110 111 112 113 114 115 116 117 118 119 120 121 122 123 124 125 126 127 128 129 130 131 132 133 134 135 136 137 138 139 140 141 142 143 144 145 146 LUCKNOW MUMBAI AHMEDABAD DELHI BANGAL ORE KOLKATA AHMEDABAD CHENNAI PUNE HYDERABAD MUMBAI KOLKATA HYDERABAD AHMEDABAD DELHI BANGALORE AHMEDABAD KANPUR KOZHIKODE KOCHI MUMBAI JAIPUR BANGALORE PUNE KOCHI KOCHI HYDERABAD BANGALORE KOLHAPUR BHUBANESHWAR BHUBANESHWAR MUMBAI CHENNAI PUNE HYDERABAD WARANGAL WARANGAL MUMBAI JALANDHAR CHENNAI TRICHY 1500416. 1500306. 63 1500152. 85 1500148 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 Page 2 0 4957494. 14 461163. 8 1164534 0. 04% 0. 75% 0. 01% 0. 59% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00%Sheet1 147 148 149 150 151 152 153 154 155 156 157 158 159 160 161 162 163 164 165 166 167 168 169 170 171 172 173 174 175 176 177 178 179 180 181 182 183 184 185 186 187 188 189 190 191 192 193 194 195 196 197 198 199 PREEYAM HIMANSHU TOLIA GOURAV TYAGI INDRAJEET SARKAR MEVISH PANCHAL PIYUSH MANOJ MODHSARAF CHANDNI JITENDRABHAI ZAVERI ABHYUDAY SINGH CHAUHAN FAIZAN KHAN KARTHIK HARIHARAN PRADEEP V SATENDER SINGH RAWAT PRAMOD AGRAWAL HARSH RANJAN ANIRBAN TARUN HAZRA KUNWAR VIKRAM SINGH BHAVIK JAGDISHBHAI BHAVSAR RAHUL VIJAYAKUMAR PRATIK MAHENDRA MANANI BHAGYASHREE SURESH PATIL SAINATH NIVLAKAR VISHAL SINGH JOHNSON BANDI ANKIT ANAND SHUBHAM JAIN ANUBHAV SINGH PADALIYA DHAVAL DEEPCHAND RADHYASHYAM GUPTA AMRITA MAZUMDAR YOGESH HARSHADBHAI PATEL SUJIT P ANUTOSH KUMAR AJEET PRATAP KHANDE NIKET CHANDAK SATYAJIT MAHAPATRO MANJUNATH RA SHREY ARORA GAURAV KAMBLE JOYDEEP DEY PANKAJ RAMESH RAI SURAJ SANJAY TAMHANE RADHE NARAYANDAS RATHI PRANAY VIJETA MUKESHKUMAR GUPTA KISHANKUMAR CHAUDHARY RAJENDER AGGARWAL SAMBHAV GUPTA RA JIV TARASHANKAR MAURYA PATHIK ASHOK DOSHI SACHIN DADABHAU ARGADE NIKUNJKUMAR BIPINCHANDRA GOR ARVIND SINGH NABA KUMAR MEDHI AFSAR TANWEER VIVA COLLEGE OF ARTS,COMMERCE AND SCIENCE ITS GHAZIABAD INSTITUTE OF ENGINEERING & MANAGEMENT BHARTIVIDYAPEETH COLLEGE OF MANAGEMENT ALKESH DINESH MODI IMS MUMBAI GHAZIABAD KOLKATA MUMBAI MUMBAI 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1500000 1499980. 58 1499898. 8 1499840. 31 1499298. 49 1499240 1498978. 6 1498975. 44 1498765. 1 1498463. 91 1498376. 77 1497879. 7 1497736 1497537. 85 1497026. 1 1496824. 5 1496650 1496525. 13 1496018. 95 1495073. 73 1493679. 6 1491854. 05 1490459 1489680 1487377. 61 1485605. 25 1484849 1483542. 5 1481977. 63 1480659. 65 1480337. 56 1480059. 8 1478627. 88 1478035. 91 1477020. 08 1476154. 24 1475551. 17 1474566. 66 1458569. 63 1455800 1450658. 24 1444595 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 00% 0. 48% -0. 01% 0. 13% -0. 05% 1. 24% 0. 00% 0. 18% 0. 08% -0. 11% 0. 54% 0. 54% 0. 68% 0. 00% 0. 50% -0. 23% 0. 73% -0. 27% 0. 16% 0. 7% -0. 04% -0. 35% 0. 13% 0. 88% -0. 38% 0. 14% 0. 12% 0. 77% -2. 62% -1. 70% 0. 95% 0. 69% 0. 23% 2. 83% -0. 67% -4. 74% -0. 41% 1. 31% -2. 50% -3. 29% -3. 72% RAJKOT R K UNIVERSITY DHARMSINGH DESAI INSTITUTE OF TECHNOLOGY & MANAGEMENT(DDIT) NADIAD NARSEEMONJI INSTITUTE OF MUMBAI MANAGEMENT STUDIES (NMIMS) INDIAN INSTITIUT OF INDORE MANAGEMENT(IIM ) VELLORE INSTITUTE OF CHENNAI MANAGEMENT (VIT) GLA UNIVERSITY II MATHURA GLA UNIVERSITY I MATHURA JAIPUR GYANVIHAR INSTITUTE DY PATIL INSTITUTE OF MANAGEMENT MUMBAI BELAPUR SWAMI VIVEKANAND INSTITUTE OF MUMBAI MANAGEMENT STUDIES CHIMANBHAI INSTITUTE OF AHMEDABAD MANAGEMENT CRESENT BUSINESS SCHOOL (B. S.ABDUR RAHMAN UNIVERSITY) CHENNAI ICFAI BUSINESS SCHOOL SANJAY GHODAWAT INSTITUTE ENTREPRENEURSHIP DEVELOPMENT INSTITUTE OF INDIA (EDI) JAIPURIA INSTITUTE OF MANAGEMENT NATIONAL INSTITUTE OF TECHNOLOGY(NIT) SCHOOL OF MANAGEMENT, KIIT UNIVERSITY INTERNATIONAL SCHOOL OF INFOMATICS & MANAGEMENT (IIIM ) TRINITY INSTITUTE OF PROFESSIONAL STUDIES NEW DELHI INSTITUTE OF MANAGEMENT (NDIM) GAHLOT INSTITUTE HERITAGE GROUP OF INSTITUTIONS GANPAT UNIVERSITY XAVIER INSTITUTE OF MANAGEMENT BIFM SOCIETY FOR EDUCATION & RESEARCH LYYOD INSTITUTE OF MANAGEMENT INDIRA COLLEGE FACULTY OF MANAGEMENT STUDIES (FMS) MS RAMAIAH COLLEGE INSTITUTE OF RURAL MANAGEMENT(IRM) ADITYA COLLEGE NSHM BUSINESS SCHOOL(NBS) UNITED BUSINESS SCHOOL SIES COLLEGE OF MANAGEMENT, NERUL D Y PATIL GREATER NOIDA INSTITUTE OF TECHNOLOGY GNVS INSTITUTE OF MANAGEMENT S V INSTITUTE OF MANAGEMENT INDIAN INSTITUTE OF MANAGEMENT(IIM) LUCKNOW AMITY BUSINESS SCHOOL DR. G D POL FOUNDATION YMT COLLEGE OF MANAGEMENT FLAME BUSINESS SCHOOL MUMBAI KOLHAPUR AHMEDABAD JAIPUR WARANGALBHUBANESHWAR JAIPUR DELHI DELHI MUMBAI KOLKATA AHMEDABAD BHUBANESHWAR DELHI DELHI PUNE DELHI BANGALORE JAIPUR MUMBAI DURGAPUR AHMEDABAD MUMBAI PUNE DELHI MUMBAI KADI DELHI AHMEDABAD MUMBAI PUNE 14 0773. 02 0 469778. 52 5920537. 72 278734. 9 1516500 0 1806549. 85 603598. 3 3727765. 43 9780 0 1359144. 05 0 427764 0 4693020. 75 4546906. 55 293725 148376. 1 2361056. 9 0 0 6257722. 15 0 0 664855. 8 24198 15104297. 55 14073842. 49 6103274. 4 10377288. 23 14504753. 51 7697288. 85 1796680. 35 19766905. 12 1469829. 15 6301258. 4 0 1486542. 2 15548000 MODERN COLLEGE PUNE NARSEEMONJI INSTITUTE OF MANAGEMENT STUDIES HYDERABAD LOVELY PROFESSIONAL UNIVERSITY JALANDHAR INDIAN INSTITUTE OF FOREIGN TRADE KOLKATA – IIFT XISS- XAVIER INSTITUTE OF SOCIAL SCIENCE RANCHI SMT.HIRABEN NANAVATI INSTITUTE OF MANAGEMENT & RESEARCH FOR WOMEN AMRITA SCHOOL OF BUSINESS SANKLP BZ COLLEGE INDIAN INSTITIUT OF MANAGEMENT(IIM) JAGANNATH INSTITUTE OF MANAGEMENT STUDIES (JIMS) VIVA COLLEGE OF MANAGEMENT JAMNALAL BAJAJ INSTITUTE OF MANAGEMENT STUDIES RAJAGIRI SCHOOL OF MANAGEMENT ST FRANCIS INSTITUTE OF MANAGEMENT MODERN INSTITUTE OF BUSINESS MANAGEMENT JAIPUR ENGINEERING COLLEGE & RESEARCH CENTRE (JECRC) SYDENHAM INSTITUTE OF MANAGEMENT STUDIES 200 201 202 203 204 205 206 207 208 NEHA ANIL SONAWANE ASHIS KUMAR NAYAK ANKUR HANUMANDASJI LOYA RAMAKRISHNA REDDY PV ASHISH VATS NILESH KALOJI PATIL CHAITANYA GANDHI ASHISH M THOMAS SAVIO IGNATIUS PEREIRA PUNE COIMBATORE PUNE BANGALORE DELHI MUMBAI MUMBAI KOCHI MUMBAI 1443019. 28 1438403. 49 1421968. 3 1403592. 6 1387933. 23 1382846. 1 1347535. 19 1315017. 23 1232887. 18 7221815. 06 6706791. 4 7983803. 6 15200000 17918166. 91 15326224 18887715. 9 14959391. 89 18177223. 5 -3. 67% -3. 27% -1. 88% -3. 44% -9. 27% -11. 30% -13. 48% -13. 75% -12. 7% Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 14-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 14-Mar2013 Disqualified NEHAL MEHUL DOSHI PUNE 2158899. 67 681523032. 99 30. 27% Disqualified INDU RAJ SHARMA JAIPUR 1746172. 62 136730223. 38 3. 01% Disqualified PARIN JAYESH JAIN MUMBAI 1689359. 21 29836586. 33 5. 38% Disqualified MRINAL AGRAWAL ICFAI BUSINESS SCHOOL GURGAON 1652256. 13 1019418. 73 9. 76% Disqualified DIPEN GIRISHBHAI DESAI SJPIM AHMEDABAD 1638720. 53 38609002. 49 5. 92% Disqualified KRATIKA KAPOOR BANASTHALI VIDYAPITH CAMPUS BANASTHALI 1602585. 74 174113219. 86 4. 03% Disqualified AKASH SINGHAL IILM DELHI DELHI 1595289. 76 Page 3 20201491. 1 0. 81% Sheet1 LAL BAHADUR SHASTRI INSTITUTE OF MANAGEMENT DELHI Disqualified as Turnover exceeded 2 crore on 14-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 rore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 14-Mar20 13 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified as Turnover exceeded 2 crore on 13-Mar2013 Disqualified SURBHI BEGWANI 1579115. 33 20629835. 15 2. 46% Disqualified HIMANSHU SHEKHAR BIT MESRA KOLKATA 1548771. 65 85329123. 21 -3. 35% Disqualified SANDEEP KUMAR INDIAN INSTITUTE OF FOREIGN TRADE DELHI (IIFT) 1513222. 8 44599450. 3 8. 38% Disqualified RAHUL SHYAMKUMAR JAIN ICFAI BUSINESS SCHOOL PUNE 1508190. 05 61504026. 95 -6. 39% Disqualified ARUN K IIT – MADRAS ENGG. DEPTCHENNAI 1501859. 94 57156690. 7 -2. 77% Disqualified DEEPANKAR KSHITIZ EMPI BUSINESS SCHOOL NALANDA INSTITUTE OF ADVANCED STUDIES DELHI 1497135. 03 69303860. 97 -2. 90% Disqualified SHEIKH HOSSAIN KOLKATA 1477135. 35 24163113. 25 -1. 52% Disqualified KUNTAL DEVESH DAVE SAL INSTITUTE OF MANAGEMENT AHMEDABAD 1447490 29854052. 5 -6. 76% Disqualified GOBIN RANA SONA COLLEGE OF MANA GMENT INSTITUTE OF MANAGEMENT & TECHNOLOGY(IMT) SALEM 1444626. 85 37832292. 25 -1. 46% Disqualified TUSHAR SHARMA MANMOHAN SINGH SIKARWAR GHAZIABAD 1380792. 05 40439282. 21 -3. 78% Disqualified ICFAI BUSINESS SCHOO L(IBS) AHMEDABAD 1064207. 96 111815542. 74 -21. 15% Page 4